Gastric mucosal lesions in chronic renal failure patients.
نویسندگان
چکیده
منابع مشابه
Gastric emptying in patients with chronic renal failure.
BACKGROUND Continuous ambulatory peritoneal dialysis (CAPD) is an alternative to the hemodialysis mode of terminal renal failure treatment. Nutritional status impairment is common among patients with end-stage renal disease, and its laboratory surrogates predict increased morbidity and mortality risk in patients on chronic haemodialysis or peritoneal dialyses. The aetiology of malnutrition is m...
متن کاملEndoscopic lesions in chronic renal failure.
To determine the frequency and type of upper gastrointestinal lesions in patients with chronic renal failure (CRF), upper G.I. endoscopy was done in 101 cases. Of the various mucosal lesions, inflammatory changes were seen maximally (40), followed by erosions (16) and ulcers (7). Other changes included atrophic gastro-duodenal folds (17), pale mucosa (11) and moniliasis (6) which was only seen ...
متن کاملGastric emptying in chronic renal failure.
BACKGROUND AND OBJECTIVES The first reference to delayed gastric emptying (GE) was made by Grodstein in 1979. Other studies have since been published, not always confirming his work. The importance of GE in anesthesia can be resumed by one of its main aspects, preoperative fasting. Delayed gastric emptying can lead to stasis and increase the risk of vomiting and aspiration. The possibility that...
متن کاملGastric acid secretion in chronic renal failure.
Contrary to the small amount of published evidence, but in accordance with clinical impression, we have found an increased incidence of peptic ulceration in people with chronic renal failure. Hyperacidic secretion in response to a standard pentagastrin test occurs in patients established on long-term dialysis treatment. The traditional liability of azotaemic patients to peptic ulceration seems ...
متن کاملParticular cutaneous lesions and chronic renal failure
A 40-year-old woman with no particular history was referred to our nephrology unit for long-standing, slowly developing chronic renal insufficiency. The blood pressure was 180/80 mmHg. Serum creatinine was 340 μmol/l and GFR 32 ml/min. Proteinuria was 0.35 G/24 h. The urinary sediment was bland. Ultrasonography disclosed two symmetrical, moderately atrophic kidneys. The diagnosis was made at fi...
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ژورنال
عنوان ژورنال: Journal of Japanese Society for Dialysis Therapy
سال: 1990
ISSN: 0911-5889,1884-6211
DOI: 10.4009/jsdt1985.23.1021